Jockology: “active rehab” for pulls and sprains

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My new Sweat Science columns are being published at www.outsideonline.com/sweatscience. Also check out my new book, THE EXPLORER'S GENE: Why We Seek Big Challenges, New Flavors, and the Blank Spots on the Map, published in March 2025.

- Alex Hutchinson (@sweatscience)

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The latest Jockology column appears in today’s Globe, dealing with the question of when “RICE” (rest, ice, compression, elevation) should turn into “MICE” (movement, ice, compression, elevation). It’s a tricky one, because there’s such a wide range of possible muscle pulls, sprains and tears that it’s difficult to give general advice. But the overall theme is that if you keep protecting and favouring a weak point for too long, you can end up harming the healing process.

The question

Ouch, I think I sprained something. How long should I stay off it?

The answer

Canadian figure skater Anabelle Langlois returned to action last month, earning a bronze medal with partner Cody Hay at a tournament in Germany one year after fracturing her fibula in a training accident. With Olympic dreams on the line, Ms. Langlois’s doctors had pursued every possible avenue in her rehabilitation, including two operations.

One thing they didn’t recommend, though, was a long period of complete rest for the injured leg. [read on…]

“Heart rate recovery” to monitor overtraining

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My new Sweat Science columns are being published at www.outsideonline.com/sweatscience. Also check out my new book, THE EXPLORER'S GENE: Why We Seek Big Challenges, New Flavors, and the Blank Spots on the Map, published in March 2025.

- Alex Hutchinson (@sweatscience)

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Back in the late 1990s, I was training under the guidance of Harry Wilson, the coach who steered Steve Ovett to Olympic gold and world records at 1,500m and the mile. Harry was an interesting mix of old-school traditionalist and cutting-edge training buff. Instead of prescribing a set amount of rest between hard intervals (like two minutes, say), he liked to wait until the athlete’s heart rate had returned to given value (generally 120bpm for me). Being a young technophile, I would wear my heart-rate monitor for these workouts in order to have instant feedback. But Harry never really trusted this newfangled technology, so I would stand there between each interval while Harry jammed his fingers into my jugular, listening to my pulse himself until it had slowed to his satisfaction. [EDIT: An astute reader points out to me that you take your pulse from the carotid artery, not the jugular vein. My apologies for any misunderstanding!]

I bring this up because, while I was browsing through the pre-prints of the Scandinavian Journal of Medicine & Science in Sports yesterday, I noticed an article by researchers at South Africa’s University of Cape Town, including Tim Noakes, on “heart rate recovery” to monitor training fatigue. The gist is as follows: 14 cyclists took part in a four-week high-intensity training program that included two interval sessions (eight repetitions of four minutes hard, with 90 seconds recovery) each week. Immediately after the final hard interval of each session, the researchers recorded how much the athlete’s heart rate decreased in the next 60 seconds.

After the four-week training period was finished, the researchers divided the subjects into two groups: those whose heart rates had recovered more and more quickly throughout the study, and those whose heart rates had recovered more and more slowly. The hypothesis was that getting better at recovery indicated the subjects were adapting to the training, while getting worse would be a sensitive indicator that they were overtraining. To test this, the subjects rode a 40-km time trial, and compared the results to a similar time trial they had ridden at the start of the study. Sure enough, the group that was recovering better rode faster, and increased power by 8.0%, compared to the slower-recovering group, which only improved power by 3.8%.

This study is part of a larger project investigating the role of heart rate recovery, so it will be interesting to see the remainder of the results when they appear. Monitoring overtraining — the failure to recover from a heavy training load, essentially — is much more of an art than a science, so having some objective tools to use would be really helpful to endurance athletes. (And I’m sure it’ll work better with heart-rate monitors than using a finger to the jugular.) Continue reading ““Heart rate recovery” to monitor overtraining”

Dynamic compression: even better than compression socks?

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My new Sweat Science columns are being published at www.outsideonline.com/sweatscience. Also check out my new book, THE EXPLORER'S GENE: Why We Seek Big Challenges, New Flavors, and the Blank Spots on the Map, published in March 2025.

- Alex Hutchinson (@sweatscience)

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normatecBack in April, when I wrote a Jockology column on compression garments, I was surprised to find out that there’s pretty good evidence that these things can actually work. “Graduated” compression socks, for instance, squeeze tightest at the bottom of the leg and gradually loosen, forcing blood back towards the heart. But regular socks have nothing on these super-fancy recovery boots that U.S. marathoner superstar Ryan Hall is now using. The picture to the right was posted on Twitter by Hall’s wife earlier this month after they started using NormaTec MVP compression boots, in this case to help recover after a long tempo run.

Basically, these things squeeze the legs with a “peristaltic pulse” that moves from the bottom of the leg towards the top, pushing blood back towards the heart. The makers refer to it as “dynamic compression” instead of the “static compression” offered by simple leggings. The idea sounds reasonable — if you believe in compression socks, then it seems like these might do the job better.

But is there any evidence, or research into how well they work? I couldn’t find any in a quick search, but I’ve e-mailed the company to see if they have anything to offer. I’ll report back when I hear from them.

Painkillers during a race or on a regular basis: a bad idea

THANK YOU FOR VISITING SWEATSCIENCE.COM!

My new Sweat Science columns are being published at www.outsideonline.com/sweatscience. Also check out my new book, THE EXPLORER'S GENE: Why We Seek Big Challenges, New Flavors, and the Blank Spots on the Map, published in March 2025.

- Alex Hutchinson (@sweatscience)

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Gretchen Reynolds of the New York Times has a really excellent article on the “prophylactic” use of “non-steroidal anti-inflammatory painkillers (NSAIDs) like ibuprofen — popping them before or during a competition, or on a regular basis before workouts, in the hopes of dulling pain or preventing subsequent soreness and swelling. It’s a must-read for everyone who does this.

In a number of studies conducted both in the field and in human performance laboratories in recent years, NSAIDs did not lessen people’s perception of pain during activity or decrease muscle soreness later… Moreover, [Indiana University researcher Stuart] Warden and other researchers have found that, in laboratory experiments on animal tissues, NSAIDs actually slowed the healing of injured muscles, tendons, ligament, and bones.

I can’t count the number of athletes I know, ranging from recreational to elite, who pop ibuprofen or equivalents on a regular “just in case” basis, hoping to avoid pain and soreness down the road. I really hope they read this article, and its conclusion:

When, then, are ibuprofen and other anti-inflammatory painkillers justified? “When you have inflammation and pain from an acute injury,” Warden says. “In that situation, NSAIDs are very effective.” But to take them “before every workout or match is a mistake.”

Ghrelin and leptin: how sleep affects your appetite

THANK YOU FOR VISITING SWEATSCIENCE.COM!

My new Sweat Science columns are being published at www.outsideonline.com/sweatscience. Also check out my new book, THE EXPLORER'S GENE: Why We Seek Big Challenges, New Flavors, and the Blank Spots on the Map, published in March 2025.

- Alex Hutchinson (@sweatscience)

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Very interesting article by Jackie Dikos in Running Times about the relationship between sleep and appetite:

There are two hormones associated with sleep that influence eating behaviors: ghrelin and leptin. Ghrelin is the hormone that lets your body know you’re hungry. Leptin’s role is to send a message to stop eating when your body has had enough. When you’re sleep-deprived, your ghrelin level increases. At the same time leptin levels decrease. So you crave additional food while simultaneously not getting the proper message to stop eating.

Seems like a pretty straightforward connection, and explains the well-documented links between getting too little sleep and gaining weight. I’ve posted before about how sleep aids athletic performance, and it’s worth adding Dikos’s conclusion:

Sleep is another way to nourish your body, just like a high-quality food choice is.